[imardmd1]

You have a link to a medical research paper that describes this mechanism involving hemoglobin breakdown?? I'd like to have it.

The usual criminal activity of the virus is the release of "a storm of cytokines." about which there is no reference to blood cells and hemoglobin.

I wonder where the mechanism you describe comes from, or how someone has discoveed that is the cause of death from the COVID virus.

[Phlyer]

a link to a medical research paper

I read it as a paper copy. The 'cytokine storm' is another disease mechanism, usually late in the cycle. But the paper said nothing about it. As I said, this is an early paper. As best I can find out, it appears that the cytokine storm is analogous to an allergic reaction where the some bodies over-react to something in the byproducts of the virus. That's not a good explanation, but it may explain why the paper said nothing about it - no hyper-allergic reactions by the time it was written.

One of the really, really ugly things about the way this whole disease was handled is that the death rate was in fact so low except among a very narrow subset of the population (primarily old, with complications from obesity, diabetes, heart disease, etc.). As a result, when an unusual response (hyper-allergic reaction) showed up, it drove the perception of the overall disease.

But for most of the deaths, the progression was to lose ability to carry oxygen in the blood, which made old, obese, heart disease, etc. die before the body could counter the virus. The best treatment - which again, didn't always work on the old, etc.) was to administer supplemental oxygen to make it easier for the remaining healthy blood cells to carry enough oxygen to keep the patient alive. The worst possible treatment was to put the patient on a ventilator - which, by forcing higher pressure air in and out of the lungs toughened up the alveoli cell membranes making it *harder* to transfer oxygen to the blood.

But, your underlying point is still important. Whether it is the breakdown of hemoglobin as the paper I saw said, or some sort of cytokine storm reaction (possibly only in hyper-allergic patients) we still did not see the disease mechanism presented, with statistical breakdowns on relation to specific additional factors (e.g. heart disease).

And then there is the whole mRNA disease-mimic approach, which is flawed on so many levels that even telling people to do it should be grounds for prison time. But my postings are too long anyway.