Actually HCQ is mentioned with approval in the introduction of this paper
"Receptor binding domains (RBD) of the viral spike play a critical role in SARS‐CoV‐2 and ACE2 fusion, so this is a very relevant target complex for viral inhibition.[ 16 ] As described above, various therapeutic strategies have been suggested to inhibit this interaction. Among some of the clinically tested drugs, chloroquine/hydroxychloroquine, arbidol, ribavirin, pensiclovir, favipiravir, nafamostat, nitazoxanide, and camostat mesylate have been shown to decrease virus–host cell binding or cellular internalization or release.
They ask for the cliff notes, and you provide.
