That's entirely too glib coming from you, when you know damn well you have no idea what the antibody-dependent enhancement (ADE) of the mRNA experimental treatments as well as the already-suspended-once AstraZeneca vaccine will be, come the winter as vectored against this ugly India variant versus the final round of any non-herd immunity host remant in the US.
As I have pointed out to you and others previously, one of the chief co-morbidities as early reported by China themselves was previously SARS/MERS vaccinations.
Late-winter 2020 in this engineered black swan event, Chinese epidemic researchers reported direct co-morbidity with any previous SARS vaccine -- which China has ("had", more likely) a population of, but only a miniscule population exists in the US -- and then China quickly shut down any further study on that, about the time they had successfully seeded their virus globally (March, 2020) and maxed out their crematoriums, ie, they stopped giving a shit about co-morbidities.
This isn't idle speculation, there are a plethora of research articles that are fully invested in the possibility of ADE in COVID-19 vaccinated folks.
"COVID-19 Vaccine Researchers Mindful of Immune Enhancement, TheScientist, May 26, 2020“With COVID-19, we have a disease which in eighty percent of people is selectively mild. So what you would not like is to give a vaccine that would not protect well and in a certain percentage of people make the disease worse.”
"Dengue remains the best-studied and one of the very few solid examples of ADE. It’s thought to occur in communities where there are multiple viral strains of dengue circulating. While antibodies against one dengue strain will typically reliably protect against that strain, things can go awry when the antibodies encounter a different strain of dengue. Instead of neutralizing the virus—that is, binding to and blocking a protein the pathogen needs to enter host cells—the antibodies only bind to the virus without neutralizing it."
" if the antibodies aren’t disabling the pathogen, they actually end up helping the virus enter macrophages to infect the cells, Trojan horse–style, explains Dennis Burton, a microbiologist at the Scripps Research Institute in California. This amplifies viral replication, potentially pushing the immune system into over-drive and paving the way for severe disease. “That’s the hallmark of ADE, basically . . . you make infection easier, you infect more cells, you get worse disease.”
"And in principle, some COVID-19 patients could develop antibodies that don’t neutralize, or produce neutralizing ones at insufficient concentrations, and then develop severe symptoms once they’re infected a second time."
Great points in your comment, but I felt compelled to make one citation of addendum to the quoted line above:
The devil in the details is the fact that there is NO PLAN on the table for 'booster' shots and they have openly admitted that they have no idea whether these shots afford any protection whatsoever beyond 6 months. That's coupled with the fact that the evidence is mounting regarding the strength of immunity from the prior infected (T-cell) vs the specific antibodies created by these chimera shots.
It doesn't take a rocket scientist to do the math pertaining to what HI might look like this winter - particularly among the 'vaccinated' - which places special emphasis upon the significance of your quoted line above (and my own comments warning of what may come this winter).
Bottom line: They don't know and, regrettably, we'll be paying the price for all of this in the end in ways I've speculated upon and hope to God I'm tin-hat city.