So the question is, is the innate immune system, in general, for adults, too weak to fight off many viruses? Because what we noticed in the original waves of the coof, is that young children hardly had any deaths. Was this because their innate immune system was stronger, and fought off the virus? Or is it the mechanism of action of the virus, is that all the intercellular signaling of the acquired immune system, goes haywire, and the associated cellular effects of inflammation, were what killed the patient?
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Wasn’t this discussed at length on the thread last summer? I don’t know if there was a conclusion. Fodder that I recall...
ACE1 binding sites (spike protein binding sites) in children are diff’t than adults, and the virus does not bind as effectively? Also, children do not have the same distribution of ACE1 sites in their nasopharyngeal mucosa (less) than do adults.
correction ACE2