Posted on 02/17/2020 2:50:09 PM PST by Vermont Lt
Continuation of daily Thread
we are steadily upping our tp reserves.
can never have too much tp!
73332
1873
2,5%
https://gisanddata.maps.arcgis.com/apps/opsdashboard/index.html#/bda7594740fd40299423467b48e9ecf6
Useless ----s.
5.56, rice, beans, water, motrin, tylenol, kaopectate, and TP
Well, we need to get more of the other stuff, to be sure.
It’s gone. The vid is down. CCP must have gotten to the guy and insist he take it down.
He was basically strolling through his “community,” which I guess you are allowed to be outside in before today, and he showed the community gate operations and how dead everything was around him.
Then, during running commentary, he said that the number of dead was likely closer to 100,000, and his wife was thinking 200,000, but he went back down to 100,000. He mentioned the fact that many of the apartments around him haven’t had people coming out, so he doesn’t know if they are alive or dead inside (paraphrased).
Pisses me off that I watched it only a hour or so ago and it is gone. Next time, I will try to grab it.
By the same reasoning:
73332
12681
17.3% survival rate.
Is it possible to get a valid positive ONE day after exposure? Don’t know, asking.
Bulk and single-cell transcriptomics identify tobacco-use disparity in lung gene expression of ACE2, the receptor of 2019-nCov
Guoshuai Cai
doi: https://doi.org/10.1101/2020.02.05.20020107
Abstract
In current severe global emergency situation of 2019-nCov outbreak, it is imperative to identify vulnerable and susceptible groups for effective protection and care. Recently, studies found that 2019-nCov and SARS-nCov share the same receptor, ACE2. In this study, we analyzed four large-scale bulk transcriptomic datasets of normal lung tissue and two single-cell transcriptomic datasets to investigate the disparities related to race, age, gender and smoking status in ACE2 gene expression and its distribution among cell types. We didn’t find significant disparities in ACE2 gene expression between racial groups (Asian vs Caucasian), age groups (>60 vs <60) or gender groups (male vs female). However, we observed significantly higher ACE2 gene expression in former smoker’s lung compared to non-smoker’s lung. Also, we found higher ACE2 gene expression in Asian current smokers compared to non-smokers but not in Caucasian current smokers, which may indicate an existence of gene-smoking interaction. In addition, we found that ACE2 gene is expressed in specific cell types related to smoking history and location. In bronchial epithelium, ACE2 is actively expressed in goblet cells of current smokers and club cells of non-smokers. In alveoli, ACE2 is actively expressed in remodelled AT2 cells of former smokers. Together, this study indicates that smokers especially former smokers may be more susceptible to 2019-nCov and have infection paths different with non-smokers. Thus, smoking history may provide valuable information in identifying susceptible population and standardizing treatment regimen.
Link for above:
https://www.medrxiv.org/content/10.1101/2020.02.05.20020107v2
I saw it on Twitter... not sure where the vid was hosted.
Yeah, I saw that several days ago. Confusing as to the racial component of susceptibility in light of other things that have been published.
It is possible that chronically breathing severe air pollution is equivalent to smoking in risk augmentation.
The only other study I am aware of is the one that had 8 samples, including 1 Asian who did smoke. This has over 200 samples.
There was an article (not scientific study) posted last night that purportedly broke down ACE2 expression by any number of racial and geographic groups. Was real weak on the origin of the data.
If the games were real, in the middle the government would come by, turn off the power, wish them luck, and weld the doors shut.
Yeah we know that cause the usual suspects from the chamber of commerce have been pooping skittles in these threads.
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