The link that silverleaf posted in 7 gives some promising discussion:
“Rosen’s team hypothesized that they could block S1P1 and the body would still fight the flu virus but without the risk of dangerous inflammation. Indeed, they found that mice treated with a drug that targeted S1P1 survived the flu without showing signs of massive cytokine production and inflammation.”
I had thought from long ago that it was not only the inflammation in the lungs but also the onslaught of white cells, presumably monos and neutrophils that created clogs upon death. So first I need to study up again on the cytokine storm stuff.
I have a lot to learn about this scenario, without a doubt.
Interesting how so much of medicine is about stopping inflammation.