Figure 9. C1q-mediated Antibody Dependent Enhancement of Ebolavirus Infection (Model). Ebola virus initiates infection by binding to its specific receptors (top panel). C1q enables binding between the virus-antibody complex and C1q ligands on the cell surface, promoting interaction between the virus and its receptor (bottom panel). Binding of the virus via the C1q molecule increases the likelihood of viral attachment to the cell surface. (Takada et al. 2003).
Ha!...I wish I was a virologist at times because these things fascinate me, but let me do my layman translation because I have been reading this stuff for weeks now.
To summarize. The virus has numerous proteins and each one has a function or they surmise it does this or that.
What the thing does, is to use the host immune system against the host and in the employ of the virus. It uses the immune reaction, comprised of the cells produces that are intended to destroy the virus for it’s own needs. It needs to break through the vascular walls as the end game, but it also need to attach it’s self..it uses the body’s own defenses to achieve what it needs to do. and by doing this it neutralizes the immune responses.
When it’s done, it breaks through the vessel walkl and leaks into its target areas which contain the cells it will feed upon.
This is when the infected become shedders and are very sick, as I explained. In fact, they may not even have a fever at this point in time. The immune system is totally gone..or compromised to such a degree that it cannot fight back.
To put it simply.