That’s the question I’ve had since last summer, and see strikingly little discussion of in such edicts:
What of those who had Covid?
I’ve seen nothing addressing the relative inoculation effects of artificial vaccination vs natural antibodies. The devotion to “get vaccinated” seems downright religious in many (seeing it’s a sufficiently advanced technology which most can’t distinguish from magic).
Since the Diamond Princess we have known a large part of the population has natural immunity.
More on T cell immunity. https://www.nature.com/articles/s41590-020-00808-x.pdf
Tcells control viral infections and provide immunological mem- orythatenableslong-lastingprotection1–3.WhereasCD4+helper T cells orchestrate the immune response and enable B cells to
produce antibodies, CD8+ cytotoxic T cells eliminate virus-infected cells. For both, recognition of viral antigens in the form of short pep- tides presented on HLAs is fundamental. In consequence, character- ization of such viral T cell epitopes4–6 is crucial for the understanding of immune defense mechanisms, but also a prerequisite for the devel- opment of vaccines and immunotherapies3,7–9.
The SARS-CoV-2 coronavirus causes COVID-19, which has become a worldwide pandemic with dramatic socioeconomic
10,11
consequences . Available treatment options are limited, and
despite intensive efforts a vaccine is so far not available. Knowledge obtained from the two other zoonotic coronaviruses SARS-CoV-1 and MERS-CoV indicates that coronavirus-specific T cell immunity is an important determinant for recovery and long-term protec- tion12–15. This T cell-mediated immune response is even more impor- tant as studies on humoral immunity to SARS-CoV-1 provided ATuRE ImmuNOLOgy
Articles
evidence that antibody responses are short-lived and can even cause or aggravate virus-associated lung pathology16,17. With regard to SARS-CoV-2, very recent studies18–20 described CD4+ and CD8+ T cell responses to viral peptide megapools in donors that had recov- ered from COVID-19 and individuals not exposed to SARS-CoV-2, the latter being indicative of potential T cell cross-reactivity21,22. The exact viral epitopes that mediate these T cell responses against SARS-CoV-2, however, were not identified and characterized in detail in these studies, but are prerequisite (1) to delineate the role of post-infectious and heterologous T cell immunity in COVID-19, (2) for establishing diagnostic tools to identify SARS-CoV-2 immunity and, most importantly, (3) to define target structures for the devel- opment of SARS-CoV-2-specific vaccines and immunotherapies. In this study, we define SARS-CoV-2-specific and cross-reactive CD4+ and CD8+ T cell epitopes in a large collection of SARS-CoV-2 con- valescent as well as nonexposed individuals and their relevance for immunity and the course of COVID-19 disease.
“That’s the question I’ve had since last summer, and see strikingly little discussion of in such edicts:”
I believe I had COVID (mild case) back in JANUARY 2020.
So I “identify as” immune from COVID.