Do you contend that epigenetic markers are not subject to mutation that can give rise to genetic variation that would be subject to selective pressure?
Do you know why some African cow herding tribes and most Northern Europeans have lactose persistence into adulthood when almost all other humans and all other mammals turn off the lactase gene after weening? Did you know that this turning off of lactose digestion after weening in mammals is due to epigenetics methylating a section proximal to the lactase gene?
So considering that all humans come from a common ancestor, how else did these epigenetic differences arise in different populations except by mutation of this epigenetic marker and natural selection that favored this trait in populations where adult milk drinking would be beneficial?
Have you forgotten who you’re talking to? We have already been all over ERVs and pseudogenes. And in both cases I have demonstrated that they do not support common descent and are much better explained in terms of Creation/ID. And no, I’m not going to start the debate all over. If you wish to refresh your memory do a search of your screenname and my screenname, combined with the term ERV or pseudogene.
==Final death rattle? Funny! The discipline of Biology has never been more productive in terms of data and beneficial products.
Any successess in biology have been made in spite of Darwin’s fairytale, not because of it.
==Do you contend that epigenetic markers are not subject to mutation that can give rise to genetic variation that would be subject to selective pressure?
What epigenetics is demonstrating is that organisms are frontloaded to match their genotype and phenotype to the selective pressures of their environment. This directly contradicts the neo-Darwinian notion that said matches are random.
==So considering that all humans come from a common ancestor, how else did these epigenetic differences arise in different populations except by mutation of this epigenetic marker and natural selection that favored this trait in populations where adult milk drinking would be beneficial?
All humans come from a common HUMAN ancestor. And I have no problem with epigenetic differences between populations. The question is, are these mutations random, or are do they reflect a purposeful, frontloaded capability to adapt to a a range of environmental pressures. I think the evidence overwhelming favors the latter.