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I wouldn't call a "dissertation of 6-fingered dwarves" as picking and choosing. It was as legitimate a choice of an example as curly wings, blue eyes/brown eyes, etc. Perhaps you would prefer if I did a comparative database of species specific genome lists.
Diabetes is no different than any other genetic condition/disorder. It doesn't operate outside the rules dominant/recessive expression. You are deliberately misinterpreting the information I am providing because it doesn't fit within your agendal thimble.
Honesty is the key to understanding.
More information on regarding lack of familial history genetic disorders, for those who wish to remain current...
the following excerpt is from
http://www.mdausa.org/publications/gen_inhr.html
How can a disease be genetic if no one else in the family has it?
This is a question often asked by people who have received a diagnosis of a genetic disorder or who have had a child with such a diagnosis. "But, doctor," they often say, "There's no history of anything like this in our family, so how can it be genetic?" This is a very understandable source of confusion.
Very often, a genetic (or hereditary) disorder occurs in a family where no one else has been known to have it.
One way for this to happen is the mechanism of recessive inheritance. In recessive disorders, it takes two mutated genes to cause disease symptoms. A single genetic mutation may have been present and passed down in a family for generations but only now has a child inherited a second mutation from the other side of the family and so developed the disease.
A similar mechanism occurs with X-linked disorders. The family may have carried a mutation on the X chromosome in females for generations, but until someone gives birth to a male child with this mutation, the genetic disorder remains only a potential, not an actual, disease. Females rarely have significant symptoms in X-linked disorders.
Another way for a child to develop a dominant or X-linked disease that's never been seen in the family follows this scenario:(bold mine) One or more of the father's sperm cells or one or more of the mother's egg cells develops a mutation. Such a mutation would never be detected by standard medical tests or even by DNA tests, which generally sample the blood cells. However, if this particular sperm or egg is used to conceive a child, he or she will be born with the mutation.
This scenario isn't rare.
Until recently, when parents who didn't have a genetic disorder and tested as "noncarriers" gave birth to a child with a genetic disorder, they were reassured that the mutation was a one-time event in a single sperm or egg cell and that it would be almost impossible for it to happen again.
Unfortunately, especially in the case of Duchenne dystrophy, this proved to be false reassurance. We now know that sometimes more than one egg cell can be affected by a mutation that isn't in the mother's blood cells and doesn't show up on standard carrier tests. Such mothers can give birth to more children with Duchenne dystrophy because subsequent egg cells with the Duchenne mutation can be used to conceive a child.
In a sense, these mothers actually are carriers - but carriers only in some of their cells. They can be thought of as "partial" carriers. Another term is mosaic carrier. It's very hard to estimate the precise risk of passing on the disorder in these cases.
It's very likely that this kind of situation occurs in other neuromuscular genetic disorders, although most haven't been as well studied as Duchenne dystrophy. For example, more than one sperm or egg cell could pass on a dominant mutation to more than one of a parent's children. Or, in a recessive disorder like spinal muscular atrophy, a child could inherit one mutation from a parent who's a full carrier, and then acquire a second genetic mutation from the other parent, a mosaic carrier. Standard carrier testing wouldn't pick up any problem in the latter parent.