Don’t forget that when the invader is a “virus”, the virus is actively attacking cells to produce these clones. The vaccine doses (which in my understanding are identical, first and second) are effectively inert, except maybe tying up some ACE2 receptors temporarily. They cannot reproduce, and are only there in order to induce the immune system to recognize and react to them. In other words, they don’t need to be ‘fought off’. If the immune system doesn’t get to some of them, they will simply degrade harmlessly.
Not really. - the lipid coat around the mRNA in the vaccine does not need to bind to a receptor site/protein to pass into the cell.
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it is possible that the substitute nucleosides are not analogs, but are different naturally modified nucleosides with control functions . They are most likely (but a guess) methylated adenosine. (But they could be less-natural analogs.)
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5167638/
Follow the link above to see details on why methylated adenosine is my best guess.
Oh this stuff works alright. Just look at the VAERS reports for all the symptoms and they are all eerily similar. All immune system malfunctions.
The doses are not the same in the double kill shots. I think the first is not that many particles injected. The second has more. If you look at VAERS you will see that most of the problems started after the 2nd injection.