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To: Republican Wildcat

The surge of third world viral vectors probably won’t matter the rate of vaccination is over a million per day now at those rates actual herd immunity will set in some time around the begins of May. Israel has already got their R0 or viral replication rate under one which by definition means the outbreak is ending for them it takes at least a R0 of one for a virus to spread. The USA has a R0 at one time above 4 we are at 1.4 and falling as the vaccines roll out. Israel has real.world data that shows at least the Pfizer version has a 94% effective rate for.asymptomatic and 96% for symptomatic that means less than 6% of a fully vaccinated population is even capable of spreading the bug as Israel R0 shows the hospital’s also reflect it 95% of people in the hospital in Israel are unvaccinated people again real.world.data with now 3 million plus vaccinated in Israel more than half the population. It’s very good news that shows conclusively that once the USA hits above 50% vaccination rates our R0’also should fall.below one and this.bug dies off for lack of hosts.


20 posted on 03/18/2021 7:40:02 PM PDT by JD_UTDallas ("Veni Vidi Vici" )
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To: JD_UTDallas

Well that’s good news. We have just about reached 1/3 now per the CDC data.


38 posted on 03/18/2021 8:04:52 PM PDT by Republican Wildcat
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To: JD_UTDallas
your realize antibodies are only 25% of the human immune defense....

So in the the worst case scenario where mutant strains dodge the mRNA vaccines, there is still:
0) spike protein antibodies
1) healthy/ tuned up immune systems
2) Memory T Cells vs ANY previous coronavirus
3) the rest of the immune defense.

when a new Flu strain shows up not everyone gets a symptomatic infection because 1) - 3) defense levels are still there.

How much of 2) Memory T Cells will hold up vs various new versions that dodge the known mRNA antibodies?
if it's not enough, then a new booster jab might be needed.

that's where a REAL problem arises called ADE which is observed in all coronaviruses (the more antibodies one gets for a coronavirus, the more severe the next infection could be)

Pathogenic priming likely contributes to serious and critical illness and mortality in COVID-19 via autoimmunity
"...Failure of SARS and MERS vaccines in animal trials involved pathogenesis consistent with an immunological priming that could involve autoimmunity in lung tissues due to previous exposure to the SARS and MERS spike protein.."

https://pubmed.ncbi.nlm.nih.gov/32292901/

in summary the wild cards are Memory T Cell defense & ADE.

to the extent the CCP bioweapons labs are still involved... they know this.
We Shall See.

46 posted on 03/18/2021 8:32:51 PM PDT by TeleStraightShooter (SARS-Cov-{Flu} infects like a flu with pRRa amino acid INSERT and it infects like a coronavirus cold)
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To: JD_UTDallas

R0 in the US has averaged well below 1 since early January


64 posted on 03/19/2021 2:12:31 AM PDT by rb22982 ( )
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