i don’t think so for a couple of reasons. first this is a problem getting oxygen across the alveolar wall (in the llungs) not a red cell capacity problem. Epo takes a while to work to start with (days to weeks) and this is not a red cell problem and too many red cells makes the blood thick/sluggish and more likely to clot. then your paper says platelets transiently increase before they decrease. in order for platelets to decrease enough to affect clotting they need to decrease by a lot - more than 50% and i’ve never seen epo drop them that significantly in chemo or dialysis patients where it is commonly used. Good thought but I don’t see this helping.
Thanks for replying. So it must have been some other mechanism in the erythropoietin treated patient if his recovery had anything to with EPO at all.