No, you haven't. Your alcohol dehydrogenase is still working just fine, converting alcohol to its aldehyde at the rate of about one drink per hour.
And yet is being overcome.
"A lot of alcohol in a short time" is layman's language, which is not precise but could be interpreted into scientificese as the consumption of close to an LD50 within one hour, a level of consumption which requires medical attention. I've yet to see anyone require medical attention from drinking just enough alcohol to get dizzy.
So dizziness is not an effect of mild or moderate alcohol toxicity - toxicity kicks in only when one requires medical attention? Sounds fishy to me.
Marijuana, on the other hand, is not water soluble. Since it is fat soluble, it very quickly goes into fat deposits throughout the body.
Where it's not binding to neuroreceptors, so having no mental effects.
that puts these chemicals in direct proximity to neuroreceptors
Is "direct proximity" the same as binding? Is there any evidence for mental effects of lipid-stored THC? As far as I know, the answers are no and no.
Nope. Unless a person has consumed so much alcohol that his life is in immediate danger without medical attention, he has not overcome his body's natural ability to destroy and remove alcohol.
So dizziness is not an effect of mild or moderate alcohol toxicity - toxicity kicks in only when one requires medical attention? Sounds fishy to me.
Only because you have not studied toxicology.
And, I think I will take this opportunity to point out that while you keep making a big deal about how feeling the effects of having a drink must be a sign of how toxic alcohol is, I can't help but notice that you are avoiding making any kind of statement about how feeling the high from marijuana must also be a sign of toxicity. Delta-9-tetrahydrocannabinol, FYI, has an LD50 about 10 times lower than alcohol. That means that it is about 10 times as toxic as alcohol, since the lower the LD50 is, the more toxic a substance is.
Is "direct proximity" the same as binding? Is there any evidence for mental effects of lipid-stored THC? As far as I know, the answers are no and no.
The fact that it is in direct proximity to a target molecule means that it is available to bind, and that it does spend a certain amount of time bound to the target. The specific amount of time it spends bound vs. not bound can only be determined by some heavy duty kinetics experiments, interpreted through complicated logarithmic functions, so I do not and cannot have the information necessary to estimate the ratio of bound/unbound THC, or to know to which molecules it is binding. In order for THC to have effects on neurological function, it is not necessary for the user to actually feel high. Those effects can occur through different pathways and affect neurological function that is not related to consciousness. For example, I do not think that hyperemesis syndrome is related to being high, but it is certainly a neurological outcome of using marijuana. In addition to receptors that are directly involved in the feeling of being high, there are thousands of other molecules in and on the cell--and what is the effect of marijuana chemicals on those other molecules? Does it affect gene expression? Does it damage DNA? Does it interfere with other metabolic pathways that are necessary for cell survival? Etc. A substance does not have to cause altered consciousness to be harmful.