[justice14]

You realize how unprovable that claim is? That is why people are upset at him. Good luck w/ that. I hope he wins, but don’t make these claims saying that calling him out on what he said is wrong.

[TigersEye]

Biology For Dummies

We should bear in mind that an acute-stress-induced surge of LH is shortly preceded by an elevation of serum progesterone from the adrenal glands. This fact suggests that such an elevation of serum progesterone may advance the secretory transformation of the endometrium resulting in embryo-endometrium asynchrony and consequently reduced chances of implantation and pregnancy if ovulation and fertilization took place.

http://www.rbej.com/content/8/1/53

“The most common causes of failure to ovulate are:
Stress, weight fluctuations, Polycystic Ovarian Syndrome (P.C.O.S.). Other causes may include disorders of the pituitary gland, thyroid gland and raised prolactin levels.”

http://cairoivf.com/treatments-ovulation_induction.html

“In conclusion, acute stress on the day of proestrus can affect female reproductive physiology. Moreover, the angiotensinergic system, through AT(1) receptors, participates in the effects of acute stress in the morning of proestrus.”

http://www.ncbi.nlm.nih.gov/pubmed/17573075

__________________________________

Animal breeding also is affected by stress. Zoos, in particular, have difficulty getting some animals to reproduce in captivity, Bentley said.

Based on animal experiments, researchers attribute much of this stress effect on sexual function to an increase in glucocorticoids - stress hormones - produced by the adrenal gland. In the brain, these glucocorticoids suppress the main reproductive hormone, GnRH, which in turn causes a shut-down of the release of the gonadotropins luteinizing hormone and follicle-stimulating hormone by the pituitary, and then a suppression of testosterone, estradiol and sexual behavior.

In 2000, however, a new reproductive hormone was discovered in birds and dubbed gonadotropin-inhibitory hormone (GnIH) because it had the opposite effect of GnRH - it inhibited release of gonadotropins, thereby suppressing reproduction.

“It’s very adaptive to not be wasting resources on reproduction during times of acute stress, to just shut down reproduction for 24 hours or so until the stress is gone,” said co-author Daniela Kaufer, a UC Berkeley assistant professor of integrative biology who looks at how stress affects molecular processes in the brain. “These functions go back in evolution a long way.”
. . ..

Kirby showed that acutely stressed rats showed increased RFRP levels for several hours, but that levels returned to normal by the next day. Chronically stressed rats, however, were left with longer-term elevations of RFRP levels in the dorsomedial hypothalamus area of the brain, and suppression of activity in the reproductive axis - the hypothalamus-pituitary-gonadal hormone cascade - that is associated with lowered sexual activity.

http://www.medicalnewstoday.com/releases/154116.php

Biology For Dummies

We should bear in mind that an acute-stress-induced surge of LH is shortly preceded by an elevation of serum progesterone from the adrenal glands. This fact suggests that such an elevation of serum progesterone may advance the secretory transformation of the endometrium resulting in embryo-endometrium asynchrony and consequently reduced chances of implantation and pregnancy if ovulation and fertilization took place.

http://www.rbej.com/content/8/1/53

“The most common causes of failure to ovulate are:
Stress, weight fluctuations, Polycystic Ovarian Syndrome (P.C.O.S.). Other causes may include disorders of the pituitary gland, thyroid gland and raised prolactin levels.”

http://cairoivf.com/treatments-ovulation_induction.html

“In conclusion, acute stress on the day of proestrus can affect female reproductive physiology. Moreover, the angiotensinergic system, through AT(1) receptors, participates in the effects of acute stress in the morning of proestrus.”

http://www.ncbi.nlm.nih.gov/pubmed/17573075

__________________________________

Animal breeding also is affected by stress. Zoos, in particular, have difficulty getting some animals to reproduce in captivity, Bentley said.

Based on animal experiments, researchers attribute much of this stress effect on sexual function to an increase in glucocorticoids - stress hormones - produced by the adrenal gland. In the brain, these glucocorticoids suppress the main reproductive hormone, GnRH, which in turn causes a shut-down of the release of the gonadotropins luteinizing hormone and follicle-stimulating hormone by the pituitary, and then a suppression of testosterone, estradiol and sexual behavior.

In 2000, however, a new reproductive hormone was discovered in birds and dubbed gonadotropin-inhibitory hormone (GnIH) because it had the opposite effect of GnRH - it inhibited release of gonadotropins, thereby suppressing reproduction.

“It’s very adaptive to not be wasting resources on reproduction during times of acute stress, to just shut down reproduction for 24 hours or so until the stress is gone,” said co-author Daniela Kaufer, a UC Berkeley assistant professor of integrative biology who looks at how stress affects molecular processes in the brain. “These functions go back in evolution a long way.”
. . ..

Kirby showed that acutely stressed rats showed increased RFRP levels for several hours, but that levels returned to normal by the next day. Chronically stressed rats, however, were left with longer-term elevations of RFRP levels in the dorsomedial hypothalamus area of the brain, and suppression of activity in the reproductive axis - the hypothalamus-pituitary-gonadal hormone cascade - that is associated with lowered sexual activity.

http://www.medicalnewstoday.com/releases/154116.php