“Akin’s answer to the question was both honest, and essentially correct, as far as the biological understanding goes.”
No, it wasn’t. With consenting adults, the pregnancy rate is about 3% at best for any one “encounter”. With a rape, it would be much less than 3%, since some rapes are without vaginal penetration, or even with that, some men don’t “finish”. So..... 1/2% maybe? That’s a guess on my part, since there is no real way of telling.
Akin said women have a biological way of stopping pregnancy from a rape, which is just not true. Pregnancy is rare, for every woman, but biology is biology, and if the lil sperm penetrates the lil egg, the woman is pregnant, period. (or lack of it)
>> “Akin said women have a biological way of stopping pregnancy from a rape, which is just not true” <<
.
If you believe that you need some education. Even an argument between husband and wife, when she is at peak fertility can prevent fertilization.
I don’t know what you’re even trying to say WRT rape, but what Akin said is largely true. the presence of the sperm and egg in the uterus does not guarantee fertilization. Stress chemically inhibits it.
http://www.rbej.com/content/8/1/53
“The most common causes of failure to ovulate are:
Stress, weight fluctuations, Polycystic Ovarian Syndrome (P.C.O.S.). Other causes may include disorders of the pituitary gland, thyroid gland and raised prolactin levels.”
http://cairoivf.com/treatments-ovulation_induction.html
“In conclusion, acute stress on the day of proestrus can affect female reproductive physiology. Moreover, the angiotensinergic system, through AT(1) receptors, participates in the effects of acute stress in the morning of proestrus.”
http://www.ncbi.nlm.nih.gov/pubmed/17573075
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Animal breeding also is affected by stress. Zoos, in particular, have difficulty getting some animals to reproduce in captivity, Bentley said.
Based on animal experiments, researchers attribute much of this stress effect on sexual function to an increase in glucocorticoids - stress hormones - produced by the adrenal gland. In the brain, these glucocorticoids suppress the main reproductive hormone, GnRH, which in turn causes a shut-down of the release of the gonadotropins luteinizing hormone and follicle-stimulating hormone by the pituitary, and then a suppression of testosterone, estradiol and sexual behavior.
In 2000, however, a new reproductive hormone was discovered in birds and dubbed gonadotropin-inhibitory hormone (GnIH) because it had the opposite effect of GnRH - it inhibited release of gonadotropins, thereby suppressing reproduction.
“It’s very adaptive to not be wasting resources on reproduction during times of acute stress, to just shut down reproduction for 24 hours or so until the stress is gone,” said co-author Daniela Kaufer, a UC Berkeley assistant professor of integrative biology who looks at how stress affects molecular processes in the brain. “These functions go back in evolution a long way.”
. . ..
Kirby showed that acutely stressed rats showed increased RFRP levels for several hours, but that levels returned to normal by the next day. Chronically stressed rats, however, were left with longer-term elevations of RFRP levels in the dorsomedial hypothalamus area of the brain, and suppression of activity in the reproductive axis - the hypothalamus-pituitary-gonadal hormone cascade - that is associated with lowered sexual activity.