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I understand that, but lactose digestion is normal(what I am saying). It is "shut down" later in life as a "normal" condition of some mammals including man(what you are saying). Lactose tolerance is "returned" by not "shutting down" the mechanism that metabolizes the sugar. If you look at the genome on gene 2 you might see something related to your answer. Again note that turning something on or off requires a mutation(change) to the mechanism coded in DNA.

LAC, 2q21

Also HYPOLACTASIA, ADULT TYPE has a good explanation of the topic

"I understand that, but lactose digestion is normal(what I am saying). It is "shut down" later in life as a "normal" condition of some mammals including man(what you are saying)."

Except that you are ignoring the fact that lactose digestion is normally shut down later in life and I am not. The system is normally more complex than you acknowledge and is disabled by mutation. Disabling complex systems would not be consistent with a goo-to-you evolution model.

"Lactose tolerance is "returned" by not "shutting down" the mechanism that metabolizes the sugar.

Au contraire, it is not 'returned'. It never left in those individual genomes where the shut-down sequence is disabled.

"If you look at the genome on gene 2 you might see something related to your answer."

Sorry, I make it a habit not to try to interpret for people. I figure that if they have something to say, they will say it.

"Again note that turning something on or off requires a mutation(change) to the mechanism coded in DNA."

What we are dealing with here is a complex system that supposedly arose (or was created) that enabled lactose-tolerance early in life and then shuts it down. That the shut-down sequence is disabled by mutation is a loss-of-function mutation. Much simpler than the turn-on sequence which required that the system be built.