Yes it is a good question.
[Acute cortical blindness: a reversible complication of acute kidney failure in a child with burns]
An 11 year old boy was admitted to the Department of Pediatrics Medical School of Vienna with 2nd and 3rd degree burns covering 30% of his body.
He presented with complications--high fever, vomiting, diarrhea and dehydration--which had led to acute renal failure.
After 6 hemodialyses renal function recovered after two weeks and the patient entered a polyuric phase. In connection with a transient dehydration the patient showed a sudden bilateral cortical blindness.
The computerized tomogram (CT) showed vague evidence of an occipital cortical ischemia. We assume that several factors have played a role in this sudden occurrence. As a result of hypovolemia and coincident anemia and electrolyte inbalance, cerebral edema and cortical tissue hypoxia with emphasis in the occipital cortical region developed in the brain possibly already damaged by burn injury. A complete reversal of the clinical state was achieved. The patient was discharged with normal vision and normalized renal function. www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=6835679&dopt=Abstract
The key elements here are the difference between "ischemia" (not enough blood flow to the area, but it isn't dead) and "neuronal loss" which means the nerves are gone.