March Report from Michigan on COVID-19

Half-Whitmer is a typical woman D’rat politician. Much is over her head.

I am originally from MI, so I’ll bite.

We will beat this thing, but here is an OS glimpse of CV-19, largely taken from another MI native, which I posted on another thread —

Some Interesting “down in the weeds” info!

Efforts began a year and a half before, and *multiple labs were involved, but in 2010, RUS/CCP Bio were successful in genetically taking the most virulent parts of the original “bat/SARS-Lk” virus, and adding it to the most virulent parts of “SARS” virus, in the Wuhan P4 genome manipulation, in which they did successfully created a NEW GENOME.

This is pages of summarized complexity by itself.

It is from thus new engineered genome VV-19 derives, and late in 2014 and in 2015, really bad stuff happened. Very quietly, RUS actually rapidly dumped significant resources into cv19 vaccine, calling it a “flu” vaccine, and for ing it on most of the population. It was generally not believed they were successful.

Already at this point, there is near impossible zoological complex union.

Analysis also reveals there was also restrictive enzyme manipulation of the HIV S component proteins, taken from glycoprotein 120 (GP120), with 4 confirmed inserts. One insert was taken from HIV-1 Gag (attached to the HIV capsid, or “nodule”), of which most bio engineers don’t understand the purpose of its insertion, and most yet believe to be mostly inert or failed. So, focus was on the other 3 HIV protein inserts. These inserts act through the CV-19 protein “spikes”, which initially look for and “plug into” the ACE-2 receptor.

However, keep in mind, the CV-19 virus is an RNA virus, and is not HIV.

That being said, it is believed the HIV S protein inserts (added/inserted to the CV-19 “nodule” spike proteins), likely serve to “fake out” the human immune system, (which doesn’t know it’s under attack), and initially even *reduces the production of *white blood cells*, enabling exponential unchecked viral growth or “load”. This means once a person really knows they are sick, and the body is trying to respond, the CV-19 virus has already been very active, and the onset of additional symptoms can be rapid.

As well, at least 35 different sequenced strands of Wuhan CV-19 have been identified around the world. One of these is known to have branched off and is called R18/G13. It is still referred to as Wuhan homogeneous, but the HIV Gag proteins are more broken down.

For now, the main receptor the CV-19 ((meaning a person has transitioned from SARS-CoV-2 into SARS/CoV-19)) RNA virus is attacking, is the Angiotensin Enzyme 2 (ACE2) recepter, (which is a Renin Aldosterone (RA) pathway), **which helps control a persons bloodpressure**!

So the CV-19 is not just attacking the lungs, it is attacking the heart, even before significantly reducing lung efficiency.

So the first steps of CV-19 virus are designed to initially attach to the ACE2 receptor, *fake out the immune system, and through cellular endocytosis exponentially replicate, while going after the RA pathway.

However, it’s not just the Wuhan Strand and the ACE2 receptor being attacked.

There are cases where CV-19 is attacking the GRP78 receptor, and the CD147 receptor.

So the spike protein on the CV-19 virus “nodule”, does already does not need to mutate, to go after other receptors. But it does have the stronger attraction or “infinity” for the bloodpressure regulating ACE2 receptor. At the same time, it can attack both GRP78 and CD147. If there is a wave 2 and wave 3, GRP78 and CD147 receptors may well be main mutated CV-19 targets.

CV-19 is likened to pre 1918 Spanish Flu in virulence, but it is not at all an influenza virus. As such while CV-19 may be 45X more virulent and robust (3 day viral life on non-porous surfaces), than H1N1, it is still not an influenza virus, and for that reason, and many other modern reasons, it is wrong to say it will kill the same percentages of people as the “Spanish Influenza”.

Part of the reason for the virulence —we have the protease called furin in our cells, and when CV-19 protein spikes have attached to an ACE2 receptor with furin near the surface, the CV-19 virus goes crazy, or “supercharged”, and this is when the CV-19 becomes roughly 1000X times as virulent.

What is written here, is largely taken from this Open Source vid — https://youtu.be/J6VEYzwSdZU

Any more than that, and increase the risk of a few people in trouble. Even in this vid, as intense as it sounds, Dr. Cottrel is being a bit careful not to get himself in trouble, so not to lose his “accesses”.

I am so on board with the hydroxychloroquine, as I have had to take much stronger related malarial drugs on deployments where up to 5 different types of malaria were present at once — As well as the use of other antibiotic therapies in the effort to reduce and even eliminate CV-19.