Posted on 11/07/2015 1:05:17 AM PST by WhiskeyX
Canary grass seed is pointed at both ends and a medium color... “parakeet seed” is usually mostly proso millet - round and light in color.
I’m going to try that pizza recipe.
Setting aside the debate over the semantics of what the word cured means with respect to diabetes, your situation may be addressed by the physicians in the videos. In general, they are saying the problem begins when the diet includes too many carbohydrates and fats in combination for too long of a time. The consequences begin with the excess carbohydrates being converted to fats and stored along with excess dietary fats in the body’s adipose tissues and throughout other areas of the body. This includes the liver, pancreas, and the insulin receptors on the cellular walls. One source is claiming the cause of insulin insensitivity is the presence of fats in the passages which transport insulin through the cellular walls.
As the liver and pancreas are increased in size by the depositions of fat, like a fattened goose, their ability to function properly becomes increasingly impaired. These impairments along with the fats causing insulin insensitivity at the cellular walls combine to reduce the effectiveness of the insulin secreted by the impaired pancreas, so the pancreas is stimulated to produce ever higher levels of glucose to remove get the cells to remove the increasing levels of glucose from the bloodstream. After a period of years, the problem worsens to the point where simply increasing the amount of insulin produced by the pancreas is insufficient to lower the blood glucose to normal levels.
This whole process is progressively worsened over time by the inability of the metabolism to burn the fats stored in the body. As the body increased the levels of insulin to compensate for the insulin insensitivity caused by the fats in the insulin receptors, the increased insulin levels adversely affected the hormones controlling the storage and burning of fats. Normally the body fasts during sleep, and the body often enters a state of ketosis that allows the fats on the body to be burned for energy to fuel the brain and the rest of the body while sleeping. When the body’s insulin levels enter a chronic state of elevation, this ketosis can no longer take place while asleep or awake. Increasingly, the metabolism fiercely conserves fats in the body and becomes more and more incapable of using the fats for fueling the metabolic processes. The end consequence is weight gain and a further worsening of the insulin insensitivity and impairment of the organs responsible for regulating these functions.
Breaking this progressive cycle requires a reversal of the increasing levels of insulin and a restoration of insulin sensitivity, according to the physicians in these videos. Anything which causes insulin levels to remain higher than normal results in the hormones regulating the storage and burning of fats in the body to continue denying the metabolism access to those stored fats. Instead, during periods of restricted caloric intake and fasting, the metabolism is forced to rely upon glucose obtained from dietary carbohydrates and protein and/or protein taken from dissolving the body’s muscles. This is why Type II diabetics typically have enlarged waistlines and have lost much of their muscles in their limbs, buttocks, and other areas of the torso. The metabolism consumed the proteins found in the muscles during periods when the high insulin levels locked away the fats to prevent them from being used as fuel for the metabolism. Accessing the stored fats requires the hormones regulating the storage and burning of those fats to be switched to burning fats, which is ketosis. For Type II diabetics, we are told this requires the insulin levels to be reduced to normal. Reducing the insulin levels to normal requires the removal of all but a minimal level of glucose from the diet, which requires the severe restriction of the carbohydrates and moderate restriction of the protein in the diet. When these two macronutrients are restricted in the diet, the only remaining macro nutrient which is available to replace them in the diet is fat. Because fat is consumed by the cells for fuel/energy as ketones without the need for insulin to do so, switching the fuel sources to fats and ketones while in a state of nutritional ketosis sets the stage for being able to reduce insulin levels. The reductions in dietary glucose permits a reduction of insulin levels, and the reduction of insulin levels in turn reduces the hormones that block the ability of the metabolism to access the fats stored in the body. The reductions in the fats stored in the body reduce the fats causing insulin insensitivity in the cellular walls, so what glucose is in the bloodstream is increasingly removed from there by the cells, improving the blood glucose levels. The increasingly restored ability to burn fats stored in the body begins to reduce the size of the fatty liver by one-third, which restores the functionality of the liver and its ability to regulate blood sugars and remove toxins from the body. The burning of the fats also removes the fats in the fatty pancreas, which improves its ability to produce insulin more and more closely to normal levels.
One of the dilemmas in this reversing the progression of Type II diabetes are the drugs used to control diabetes, Some, not all, diabetic drugs cause the pancreas to secrete more insulin in an effort to compensate for the cells’ insulin insensitivity. Unfortunately, such drugs worsen the inability of the metabolism to burn the fats stored in the body, just as the increased natural secretions of insulin do. So long as the metabolism is being blocked from using these stored fats as fuel, the ability to remove the fats from the cellular walls, liver, and pancreas will be impaired. On the other hand, the failure to use the diabetic medications which increase insulin levels risks high blood glucose levels which can be very harmful in the short term and/or long term. So, this dilemma leads to the question of what a Type II diabetic person can do when both courses of action may ultimately be very harmful? Some physicians have suggested using an alternative type of diabetic medication which does not work by increasing insulin levels. Unfortunately, these drugs may or may not have their own dangerous side effects, including kidney damage, heart problems, and so forth.
Another approach has been suggested by a medical study which claims better long term outcomes resulted when diabetic medications were not used to more tightly control blood glucose levels. They suggested looser control of blood glucose levels and setting a minimum blood glucose level in addition to the high blood glucose level. The thinking behind this recommendation was that the tight control of blood glucose levels were achieved by using higher levels of insulin, which worsened the inability to burn stored fats, accelerated weight gains, and resulted in earlier progressions of the diabetes to its conclusion in death.
To the extent the physicians in the videos and the studies may be correct, the challenge for a person with Type II diabetes is to discover in some way how to reverse the progression of the diabetes, reduce insulin levels to burn fat and lose weight, and do so without incurring unacceptable risks from using or not using diabetic medications and/or exogenous insulin.
In your case, the use of the glyburide may mean your tight control of the blood glucose levels is simply forcing the liver to store the glucose as fats on the liver and elsewhere, which progressively worsens the conditions as the months and years go by, until the ability to compensate enough to maintain control is lost. This is the typical progression of the disease. According to these physicians in the videos, the only way to stop the progression of the diabetes is to remove the fats from the liver, pancreas, insulin receptors of the cells, and rest of the body.
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