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Do you hear that? LOW SODIUM LEVELS MIGHT BE DANGEROUS TO YOUR HEART......

Just another reason to ignore the Obamas, the mainstream media, and the rest of the liberal establishment.

1 posted on 01/19/2015 9:53:30 AM PST by SoFloFreeper
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To: SoFloFreeper

Tells me that these “studies” don’t have any credibility, and should be ignored.


87 posted on 01/19/2015 11:14:40 AM PST by I want the USA back (Media: completely irresponsible. Complicit in the destruction of this country.)
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To: SoFloFreeper
The doctor's surname, Kalogeropoulos, is interesting. It appears as if he had an ancestor who was the son of a monk.

Maybe it was a monk who joined the monastery after his wife died.

88 posted on 01/19/2015 11:17:18 AM PST by Verginius Rufus
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To: SoFloFreeper

Just be sure to eat from the basic food groups — grease, salt, caffeine, sugar, and preservatives.


91 posted on 01/19/2015 11:24:59 AM PST by Bob (Violence in islam? That's not a bug; it's a feature.)
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To: SoFloFreeper
You mean there was no deep fat? No steak or cream pies or... hot fudge?
94 posted on 01/19/2015 11:27:33 AM PST by Rodamala
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To: SoFloFreeper

My favorite salt is coarse granule sea salt. Goes great with salads!


99 posted on 01/19/2015 11:36:56 AM PST by PJ-Comix (Coakley/Gruber 2016!!!)
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To: SoFloFreeper

That’s why I never altered my relatively high usage of salt.


107 posted on 01/19/2015 11:49:35 AM PST by TigersEye (ISIS is the tip of the spear. The spear is Islam.)
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To: SoFloFreeper

Low salt can cause electrolytes to go haywire, especially in those taking diuretics. Some people need more salt than others. We pretty much ignore recommendations and salt to taste even taking salt tablets when we have sweated a lot. Mother in law has been a salt Nazi for years and has gotten too low several times, collapsing with no energy. We tell her to have them test for low sodium and sure enough she is in trouble. But when she starts feeling good, she goes back to the salt restriction. Swelling in her lower legs worries her, as I can understand but experience should teach her. Too much pop culture medicine advice on TV I guess.


109 posted on 01/19/2015 12:06:48 PM PST by outinyellowdogcountry
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To: SoFloFreeper

I don’t hear top conservatives, the conservative media or any conservative entities saying anything different about salt.

So this has NOTHING to do wuth Conservative v Liberal, I’d say.


111 posted on 01/19/2015 12:13:07 PM PST by BunnySlippers (I LOVE BULL MARKETS . . .)
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To: SoFloFreeper

I eat a lot of salt and my blood pressure is normal.


112 posted on 01/19/2015 12:16:25 PM PST by Fresh Wind (The last remnants of the Old Republic have been swept away)
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To: SoFloFreeper

I’ve been posting this for years now, it first appeared in, of all places, The New York Times.

If you have heart disease, or if you DON’T have heart disease and don’t want to get it, this is worth your time to read.
+++

By GARY TAUBES
Published: June 2, 2012

Oakland, Calif.

THE first time I questioned the conventional wisdom on the nature of a healthy diet, I was in my salad days, almost 40 years ago, and the subject was salt. Researchers were claiming that salt supplementation was unnecessary after strenuous exercise, and this advice was being passed on by health reporters. All I knew was that I had played high school football in suburban Maryland, sweating profusely through double sessions in the swamp like 90-degree days of August. Without salt pills, I couldn’t make it through a two-hour practice; I couldn’t walk across the parking lot afterward without cramping.

While sports nutritionists have since come around to recommend that we should indeed replenish salt when we sweat it out in physical activity, the message that we should avoid salt at all other times remains strong. Salt consumption is said to raise blood pressure, cause hypertension and increase the risk of premature death. This is why the Department of Agriculture’s dietary guidelines still consider salt Public Enemy No. 1, coming before fats, sugars and alcohol. It’s why the director of the Centers for Disease Control and Prevention has suggested that reducing salt consumption is as critical to long-term health as quitting cigarettes.

And yet, this eat-less-salt argument has been surprisingly controversial — and difficult to defend. Not because the food industry opposes it, but because the actual evidence to support it has always been so weak.

When I spent the better part of a year researching the state of the salt science back in 1998 — already a quarter century into the eat-less-salt recommendations — journal editors and public health administrators were still remarkably candid in their assessment of how flimsy the evidence was implicating salt as the cause of hypertension.

“You can say without any shadow of a doubt,” as I was told then by Drummond Rennie, an editor for The Journal of the American Medical Association, that the authorities pushing the eat-less-salt message had “made a commitment to salt education that goes way beyond the scientific facts.”

While, back then, the evidence merely failed to demonstrate that salt was harmful, the evidence from studies published over the past two years actually suggests that restricting how much salt we eat can increase our likelihood of dying prematurely. Put simply, the possibility has been raised that if we were to eat as little salt as the U.S.D.A. and the C.D.C. recommend, we’d be harming rather than helping ourselves.

WHY have we been told that salt is so deadly? Well, the advice has always sounded reasonable. It has what nutritionists like to call “biological plausibility.” Eat more salt and your body retains water to maintain a stable concentration of sodium in your blood. This is why eating salty food tends to make us thirsty: we drink more; we retain water. The result can be a temporary increase in blood pressure, which will persist until our kidneys eliminate both salt and water.

The scientific question is whether this temporary phenomenon translates to chronic problems: if we eat too much salt for years, does it raise our blood pressure, cause hypertension, then strokes, and then kill us prematurely? It makes sense, but it’s only a hypothesis. The reason scientists do experiments is to find out if hypotheses are true.

In 1972, when the National Institutes of Health introduced the National High Blood Pressure Education Program to help prevent hypertension, no meaningful experiments had yet been done. The best evidence on the connection between salt and hypertension came from two pieces of research. One was the observation that populations that ate little salt had virtually no hypertension. But those populations didn’t eat a lot of things — sugar, for instance — and any one of those could have been the causal factor. The second was a strain of “salt-sensitive” rats that reliably developed hypertension on a high-salt diet. The catch was that “high salt” to these rats was 60 times more than what the average American consumes.

(Page 2 of 2)

Still, the program was founded to help prevent hypertension, and prevention programs require preventive measures to recommend. Eating less salt seemed to be the only available option at the time, short of losing weight. Although researchers quietly acknowledged that the data were “inconclusive and contradictory” or “inconsistent and contradictory” — two quotes from the cardiologist Jeremiah Stamler, a leading proponent of the eat-less-salt campaign, in 1967 and 1981 — publicly, the link between salt and blood pressure was upgraded from hypothesis to fact.

In the years since, the N.I.H. has spent enormous sums of money on studies to test the hypothesis, and those studies have singularly failed to make the evidence any more conclusive. Instead, the organizations advocating salt restriction today — the U.S.D.A., the Institute of Medicine, the C.D.C. and the N.I.H. — all essentially rely on the results from a 30-day trial of salt, the 2001 DASH-Sodium study. It suggested that eating significantly less salt would modestly lower blood pressure; it said nothing about whether this would reduce hypertension, prevent heart disease or lengthen life.

While influential, that trial was just one of many. When researchers have looked at all the relevant trials and tried to make sense of them, they’ve continued to support Dr. Stamler’s “inconsistent and contradictory” assessment. Last year, two such “meta-analyses” were published by the Cochrane Collaboration, an international nonprofit organization founded to conduct unbiased reviews of medical evidence. The first of the two reviews concluded that cutting back “the amount of salt eaten reduces blood pressure, but there is insufficient evidence to confirm the predicted reductions in people dying prematurely or suffering cardiovascular disease.” The second concluded that “we do not know if low salt diets improve or worsen health outcomes.”

The idea that eating less salt can worsen health outcomes may sound bizarre, but it also has biological plausibility and is celebrating its 40th anniversary this year, too. A 1972 paper in The New England Journal of Medicine reported that the less salt people ate, the higher their levels of a substance secreted by the kidneys, called renin, which set off a physiological cascade of events that seemed to end with an increased risk of heart disease. In this scenario: eat less salt, secrete more renin, get heart disease, die prematurely.

With nearly everyone focused on the supposed benefits of salt restriction, little research was done to look at the potential dangers. But four years ago, Italian researchers began publishing the results from a series of clinical trials, all of which reported that, among patients with heart failure, reducing salt consumption increased the risk of death.

Those trials have been followed by a slew of studies suggesting that reducing sodium to anything like what government policy refers to as a “safe upper limit” is likely to do more harm than good. These covered some 100,000 people in more than 30 countries and showed that salt consumption is remarkably stable among populations over time. In the United States, for instance, it has remained constant for the last 50 years, despite 40 years of the eat-less-salt message. The average salt intake in these populations — what could be called the normal salt intake — was one and a half teaspoons a day, almost 50 percent above what federal agencies consider a safe upper limit for healthy Americans under 50, and more than double what the policy advises for those who aren’t so young or healthy. This consistency, between populations and over time, suggests that how much salt we eat is determined by physiological demands, not diet choices.

One could still argue that all these people should reduce their salt intake to prevent hypertension, except for the fact that four of these studies — involving Type 1 diabetics, Type 2 diabetics, healthy Europeans and patients with chronic heart failure — reported that the people eating salt at the lower limit of normal were more likely to have heart disease than those eating smack in the middle of the normal range. Effectively what the 1972 paper would have predicted.

Proponents of the eat-less-salt campaign tend to deal with this contradictory evidence by implying that anyone raising it is a shill for the food industry and doesn’t care about saving lives. An N.I.H. administrator told me back in 1998 that to publicly question the science on salt was to play into the hands of the industry. “As long as there are things in the media that say the salt controversy continues,” he said, “they win.”

When several agencies, including the Department of Agriculture and the Food and Drug Administration, held a hearing last November to discuss how to go about getting Americans to eat less salt (as opposed to whether or not we should eat less salt), these proponents argued that the latest reports suggesting damage from lower-salt diets should simply be ignored. Lawrence Appel, an epidemiologist and a co-author of the DASH-Sodium trial, said “there is nothing really new.” According to the cardiologist Graham MacGregor, who has been promoting low-salt diets since the 1980s, the studies were no more than “a minor irritation that causes us a bit of aggravation.”

This attitude that studies that go against prevailing beliefs should be ignored on the basis that, well, they go against prevailing beliefs, has been the norm for the anti-salt campaign for decades. Maybe now the prevailing beliefs should be changed. The British scientist and educator Thomas Huxley, known as Darwin’s bulldog for his advocacy of evolution, may have put it best back in 1860. “My business,” he wrote, “is to teach my aspirations to conform themselves to fact, not to try and make facts harmonize with my aspirations.”


116 posted on 01/19/2015 12:43:00 PM PST by Balding_Eagle (The Gruber Revelations are proof that God is still smiling on America.)
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To: SoFloFreeper

Salt is cheap, tastes good, goes on anything, and essential for life; no wonder the government hates it.


117 posted on 01/19/2015 12:55:19 PM PST by WMarshal (Free citizen, never a subject or a civilian)
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To: SoFloFreeper

I spent a few summers working in aluminum reduction plants in the 70s. Standard uniform was a full set of cotton long johns, jeans, and long sleeve flannel shirt. When the hot bath would splash on you or your jeans would catch fire, the soaking wet long johns would give you a mild steam burn instead of charred flesh. In the break room and all over the plant were the cardboard boxes restaurants get that are full of the little salt packs. We were encouraged to eat plenty a day, 8 or 10 weren’t uncommon. My first day at lunch I saw an old timer pour 4 salts into his coke can and drink it as he ate. My first week I was doing the same. When summer was over I left the job and quit the salt with no bad effects.


121 posted on 01/19/2015 1:13:23 PM PST by eartrumpet
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To: SoFloFreeper
A few twists and turns to this. First of all, it is common knowledge that low blood sodium can be bad for you, because it is an essential electrolyte. In physiology, the primary ions of electrolytes are sodium (Na+), potassium (K+), calcium (Ca2+), magnesium (Mg2+), chloride (Cl−), hydrogen phosphate (HPO42−), and hydrogen carbonate (HCO3−). Electrolytes exist in a balance with each other, so too much or too little of any of them can be a problem. The kidneys are in charge of eliminating excess electrolytes. Now this being said, table salt, sodium chloride, is typically good because it contains two of the electrolytes. However, importantly, it is not the only form of sodium out there. A real winner for "high blood pressure in a bag" is Chex Mix, normally consumed around the holidays. This is because it contains no fewer than FIVE different kinds of sodium. Table salt (Sodium chloride) Baking soda (Sodium bicarbonate) Trisodium phosphate Disodium inosinate Disodium guanylate However, there are many more commonly used sodium food additives, like: Monosodium glutamate (MSG) Sodium nitrite Sodium nitrate Sodium saccharine Sodium benzoate Sodium aluminosilicate Sodium iodide Sodium iodate Sodium ferrocyanide (anti-caking agent) Sodium fluoride So, properly speaking, before they say that sodium is safe or unsafe at given levels, they need to test each of these forms of sodium individually and in groups. I doubt they have done this.
123 posted on 01/19/2015 1:36:40 PM PST by yefragetuwrabrumuy ("Don't compare me to the almighty, compare me to the alternative." -Obama, 09-24-11)
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To: VeniVidiVici

bump


130 posted on 01/19/2015 7:57:24 PM PST by VeniVidiVici ( Better a conservative teabagger than a liberal teabagee)
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