1. Ebola survives in the cold and certain surfaces.
That is strictly laboratory data, measured in controlled conditions, and I have discussed that at length in other posts. If you want to know whether Ebola viruses are stable in situations where someone could actually be infected, you have to study the virus in those conditions.
==== Life is not controlled, and the temperature
data, and binding-survival data is relevant.
You are sophomoric to assume the virus will not mutate.
2. And it survives MONTHS in humans who are allegedly “cured”.
It is known that virus takes weeks to clear from some bodily fluids. ... But all patients had completely eliminated virus after 400 days. Obviously, this is a potential means of transmission.
======= Thank you. Q.E.D.
3. And you have ignored the animal reservoirs which are about to develop and grow.
The virus has always been in an animal reservoir, probably bats. It is not about to “develop and grow”;
====== “Probably” means you do not KNOW the extent
of the animal reservoir. And NO ONE knows how many
animals species it will survive in, on the N. America
continent (yet).
4. And where is the data showing filoviruses are “larger” than the droplets. That is nonsense.
A filovirus is 80 nm in diameter and anywhere from about 900 to 14,000 nm, or 0.9 to 14 micrometers in length. Airborne particles generated by sneezing, coughing, etc., are smaller than about 5-10 micrometers. The average size of such particles is smaller. ... So, even if virus were present in respiratory secretions (if they contain blood, virus could be present), very few virions would fit in a particle.
============= Rain drops are between 1 and 50 microns in
diameter. Nebulized aerosol droplets are 2 to 20 microns.
The filamentous Ebola, stretched out which it need not be,
is 970 nm which is 0.9 microns. Therefore, Q.E.D.
Reston 1989 Ebola outbreak. This involved a monkey quaratine facility and the virus spread from room to room through the air. This is not the same strain of Ebola that has our attention now.
“Of course I believed it,” he said. “I was shocked. It was hundreds of monkeys involved right there in Reston.”
He said everyone’s mind turned to the full weight of what was happening. What if Ebola got into the general population? Were the handlers infected? And just as troubling: Why were monkeys in another contained room dying? Had the virus mutated? Was it now being transmitted through the air?
That is strictly laboratory data, measured in controlled conditions, and I have discussed that at length in other posts. If you want to know whether Ebola viruses are stable in situations where someone could actually be infected, you have to study the virus in those conditions.
==== Life is not controlled, and the temperature data, and binding-survival data is relevant. You are sophomoric to assume the virus will not mutate.
That is the point. It has only been shown to survive for prolonged periods in controlled environments--in environments that do not assault the virus with factors like temperature, humidity, changes in pH, environmental chemicals, etc. Biological entities typically are not very durable in uncontrolled environments.
Also, I *assume* that the virus mutates. It would be extremely unusual if it did NOT mutate, since viruses, especially RNA viruses, have very unstable genetic material. They cannot mutate in such a way that they change their shape or the functions of their proteins too much, because then, they would be unable to cause infections--but, within limits, they mutate a lot.
“Probably” means you do not KNOW the extent of the animal reservoir. And NO ONE knows how many animals species it will survive in, on the N. America continent (yet).
Actually, "probably" means that everyone is pointing fingers at bats, but nothing has been proven. The evidence is all circumstantial.
Ebola causes different symptoms in different animals. Dogs don't get symptoms, but they have been shown to get infected. Pigs get a respiratory illness, and they can spread the disease through respiratory droplets (they sneeze a lot, contaminating their environment). But they don't die (at least in lab studies). And so on. Many viruses cause mild illness in bats, but are deadly to humans. No one can possibly know how the virus will affect a species without testing it.
Rain drops are between 1 and 50 microns in diameter. Nebulized aerosol droplets are 2 to 20 microns. The filamentous Ebola, stretched out which it need not be, is 970 nm which is 0.9 microns. Therefore, Q.E.D.
The 970 nm length is a minimum; it is up to 14,000 nm long. And it can't be "stretched"--it might coil or not, but its volume is still the same. And it fits best in the large particles that fall quickly to the ground. I doubt that the small particles can hold an infectious dose, and they dry so quickly that the virus wouldn't survive anyway.
Nebulizing, as in laboratory experiments, is not really pertinent to a natural setting. For aerosol studies, the virus is mechanically nebulized directly into an animals face. Humans don't nebulize... Some medical procedures can generate aerosols or droplets.