Posted on 04/09/2020 8:24:37 PM PDT by FreedomPoster
Thanks. I didn’t know. Maybe if plasma donations help at all, they would be for the less critical patients.
It really seems like if possible prevention could be the key to not letting it damage the lungs so much. There seems to be evidence that vitamin d deficiency plays toward a worse case. And if anyone has any metabolic issues, even just obesity, stop eating sugar and breads cold turkey (which is hard as the cravings will come - but add your favorite salty, non manufactured treats like bacon or veggies and dips).
Thanx
His latest video is pretty good, too. It’s about what may be going on with the lungs and the odd pneumonia.
https://onlinelibrary.wiley.com/doi/epdf/10.1002/jmv.25839
That paper about recovery in a seriously ill patient being treated with erythropoietin kept me wondering about what might be happening with that.
I ran across this:
https://content.iospress.com/articles/advances-in-neuroimmune-biology/nib140076
“If high dose of recombinant human erythropoietin is used, it will stimulate platelet production, which is a transient effect. Expanded erythropoiesis exerts a negative impact on platelet production.”
Wouldn’t that be what you’d want in COVID-19 patients who need oxygen (more red blood cells) but are having clotting in the small vessels of the lungs (less platelets)? Would the EPO given to the COVID-19 patient who recovered be considered high dose?
I probably don’t know what I’m talking about but I thought I’d run the idea by an MD.
Just wanted to direct you to post #124 in this thread.
http://freerepublic.com/focus/news/3833628/posts?page=124#124
i don’t think so for a couple of reasons. first this is a problem getting oxygen across the alveolar wall (in the llungs) not a red cell capacity problem. Epo takes a while to work to start with (days to weeks) and this is not a red cell problem and too many red cells makes the blood thick/sluggish and more likely to clot. then your paper says platelets transiently increase before they decrease. in order for platelets to decrease enough to affect clotting they need to decrease by a lot - more than 50% and i’ve never seen epo drop them that significantly in chemo or dialysis patients where it is commonly used. Good thought but I don’t see this helping.
I just saw gas_dr’s post at #102. So erythropoietin takes too long for what I was thinking about in #124. Never mind.
Thanks for replying. So it must have been some other mechanism in the erythropoietin treated patient if his recovery had anything to with EPO at all.
I’would think so
Saw that yesterday. We have a mystery that is slowly being sorted out. I am going with Zelenko’s suggestion, treat any who present in early stage with an aggressive dosing of at least gydroxychloroquine plus zinc. Once a patient is close to benting the disease may be too strong to treat with HCQQ and zinc alone. Treat the disease early and aggressively seems most prudent. HCQ and zinc will be crucial to getting America opened back up, sooner rather than later. Damn the globalist bastards whom Fouci represents.
I'm not a doctor, but am deeply interested in this thread. MHGinTN, I believe you are on target. The disagreements here may be related to when a patient is treated. Everyone may be correct. The regimen for the outpatient may be quite different than needed in the more advanced stage according to damage already done. Has anyone kept up with Israel's new anti-inflammatory / immunomodulator drug? I like the approach for advanced cases.
Please, everyone, stay cool. This is a great thread and I greatly appreciate what everyone is contributing. BTW, Ukraine is going to have 8 million people on Dr Z's HCQ/Zn regimen, mainly as a prophylaxis.
KY not TN:-)
I ran across this. Don’t know if it’s any value to you but i thought i’d pass it along.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7128154/pdf/main.pdf
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