I’ve been doing a lot more research, and have narrowed down the particular species of enterobacter which may be mostly to blame. E. cloacae. This is what they focused on in the experiment, and it would have been nice if they had said so, outside of the professional journals.
Granted, while reducing the number of other Enterobacteriaceae family in the flora can prevent a lot of other nasty, opportunistic infections, especially those in the enterobacter genus, E. cloacae seems to be the real villain for overweight, here.
To make matters worse, it is also adept at developing antibiotic resistance.
Fortunately, there are several OTC supplements that really put the blocks to it and a few other nasty bacteria, the most favored of which is Caprylic acid, found in coconut oil.
While Caprylic acid is sold OTC by itself, virgin coconut oil is regarded as more effective.
There were several reports of people with severe infections who consumed the typical max dose of coconut oil (2 tbsp/3x daily), and got flu-like symptoms typical of a major bacterial fight, including, as they said, some extremely foul smelling vomiting and diarrhea, along with copious amounts of gas for the better part of a week before they adapted to their new floral balance.
Fortunately, this only seems to happen with a preexisting major floral imbalance. Those who have some sensitivity start with a tsp. a day with food, and work their way up.
Because below 76F, coconut oil is solid, like butter, it can be somewhat difficult to consume, but by my own experience, it blends well up to maximum dosage with warmed Atkins drink. Which would appeal on a low carb diet.